Laboratory of Neurodegenerative Diseases and Aging57
Our four major questions are:
1. What is the "cellular basis" of neurodegenerative diseases?
2. How can we ameliorate the toxicity of aggregated proteins associated with neurodegenerative diseases?
3. What's the relationship between neural cellular aging and late-onset neurodegenerative diseases?
4. What's the physiological consequence of altered cellular ion & mRNA homeostasis?
Advisor Professor : Lee, Sung-Bae
SB LAB(Lab of Neurodegenerative diseases and Aging) Homepage
1. What is the "cellular basis" of neurodegenerative diseases?
2. How can we ameliorate the toxicity of aggregated proteins associated with neurodegenerative diseases?
3. What's the relationship between neural cellular aging and late-onset neurodegenerative diseases?
4. What's the physiological consequence of altered cellular ion & mRNA homeostasis?
Advisor Professor : Lee, Sung-Bae
SB LAB(Lab of Neurodegenerative diseases and Aging) Homepage
- Date issued
- 25 2020 - 2024
- 30 2013 - 2019
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Recent Submissions
RSS 1.0
RSS 2.0
ATOM 1.0
- Monitoring lipid alterations in Drosophila heads in an amyotrophic lateral sclerosis model with time-of-flight secondary ion mass spectrometry
- A comparison study of pathological features and drug efficacy between Drosophila models of C9orf72 ALS/FTD
- CBP-Mediated Acetylation of Importin alpha Mediates Calcium-Dependent Nucleocytoplasmic Transport of Selective Proteins in Drosophila Neurons
- Brain Endothelial Cells Utilize Glycolysis for the Maintenance of the Transcellular Permeability
- Peroxiredoxin 3 deficiency induces cardiac hypertrophy and dysfunction by impaired mitochondrial quality control