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Showing results 1 to 4 of 4

Article
A missense allele of KARRIKIN-INSENSITIVE2 impairs ligand-binding and downstream signaling in Arabidopsis thaliana
  • Lee, In Hye ; 
  • Kim, Kug Lae ; 
  • Lee, Su Min ; 
  • Lee, Seung Jun ; 
  • Hwang, Eun Jin ; 
  • Shin, Ki Hye ; 
  • Kim, Da Young ; 
  • Choi, Jung Ki ; 
  • Choi, Hyun Mo ; 
  • Cha, Jeong Seok ; 
    • et al
  • 2018-07
  • Lee, In Hye. (2018-07). A missense allele of KARRIKIN-INSENSITIVE2 impairs ligand-binding and downstream signaling in Arabidopsis thaliana. doi: 10.1093/jxb/ery164
  • Oxford University Press
  • View : 601
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Article
Antagonistic Roles of PhyA and PhyB in Far-Red Light-Dependent Leaf Senescence in Arabidopsis thaliana
  • Lim, Junhyun ; 
  • Park, Ji-Hwan ; 
  • Jung, Sukjoon ; 
  • Hwang, Daehee ; 
  • Nam, Hong Gil ; 
  • Hong, Sunghyun
  • 2018-09
  • Lim, Junhyun. (2018-09). Antagonistic Roles of PhyA and PhyB in Far-Red Light-Dependent Leaf Senescence in Arabidopsis thaliana. doi: 10.1093/pcp/pcy153
  • Oxford University Press
  • View : 960
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Article
iNID: An Analytical Framework for Identifying Network Models for Interplays among Developmental Signaling in Arabidopsis
  • Choi, Daeseok ; 
  • Choi, Jaemyung ; 
  • Kang, Byeongsoo ; 
  • Lee, Seungchul ; 
  • Cho, Young-hyun ; 
  • Hwang, Ildoo ; 
  • Hwang, Daehee
  • 2014-05
  • Choi, Daeseok. (2014-05). iNID: An Analytical Framework for Identifying Network Models for Interplays among Developmental Signaling in Arabidopsis. Molecular Plant, 7(5), 792–813. doi: 10.1093/mp/sst173
  • Cell Press
  • View : 806
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Article
Methionine synthase 1 provides methionine for activation of the GLR3.5 Ca2+ channel and regulation of germination in Arabidopsis
  • Ju, Chuanli ; 
  • Kong, Dongdong ; 
  • Lee, Yuree ; 
  • Ge, Gege ; 
  • Song, Yanan ; 
  • Liu, Jiawen ; 
  • Kwak, June Myoung
  • 2020-01
  • Ju, Chuanli. (2020-01). Methionine synthase 1 provides methionine for activation of the GLR3.5 Ca2+ channel and regulation of germination in Arabidopsis. Journal of Experimental Botany, 71(1), 178–187. doi: 10.1093/jxb/erz431
  • Oxford University Press
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