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Showing results 1 to 6 of 6

  • Noh, Byeongju ; 
  • Lee, Hyun-Ju ; 
  • Lee, Jiyun ; 
  • Lee, Ji-Eun ; 
  • Joo, Bitna ; 
  • Jung, Young-Hun ; 
  • Park, Minwoo ; 
  • Kang, Sora ; 
  • Oh, Seokjun ; 
  • Hwang, Jeong-Woo ; 
  • et al
  • 2025-10
  • ACS Biomaterial Science & Engineering, v.11, no.11, pp.6710 - 6726
  • American Chemical Society
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Idebenone Decreases Aβ Pathology by Modulating RAGE/Caspase-3 Signaling and the Aβ Degradation Enzyme NEP in a Mouse Model of AD

  • Lee, Hyun-ju ; 
  • Jeong, Ha-Ram ; 
  • Park, Jin-Hee ; 
  • Hoe, Hyang-Sook
  • 2021-09
  • Lee, Hyun-ju. (2021-09). Idebenone Decreases Aβ Pathology by Modulating RAGE/Caspase-3 Signaling and the Aβ Degradation Enzyme NEP in a Mouse Model of AD. Biology, 10(9). doi: 10.3390/biology10090938
  • MDPI
  • View : 550
  • Download : 98
  • 2021-06
  • Cho, Sukhee. (2021-06). Impact of Genetic Risk Factors for Alzheimer’s Disease on Brain Glucose Metabolism. Molecular Neurobiology, 58(6), 2608–2619. doi: 10.1007/s12035-021-02297-x
  • Springer
  • View : 688
  • Download : 0

Regorafenib Regulates AD Pathology, Neuroinflammation, and Dendritic Spinogenesis in Cells and a Mouse Model of AD

  • Han, Kyung-Min ; 
  • Kang, Ri Jin ; 
  • Jeon, Hyongjun ; 
  • Lee, Hyun-ju ; 
  • Lee, Ji-Soo ; 
  • Park, HyunHee ; 
  • Jeon, Seong Gak ; 
  • Suk, Kyoungho ; 
  • Seo, Jinsoo ; 
  • Hoe, Hyang-Sook
  • 2020-07
  • Han, Kyung-Min. (2020-07). Regorafenib Regulates AD Pathology, Neuroinflammation, and Dendritic Spinogenesis in Cells and a Mouse Model of AD. doi: 10.3390/cells9071655
  • MDPI AG
  • View : 754
  • Download : 245
  • 2017-01
  • BMB Reports, v.50, no.1, pp.5 - 11
  • The Biochemical Society of the Republic of Korea
  • View : 935
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  • Lee, Hyun-ju ; 
  • Woo, Hanwoong ; 
  • Lee, Ha-Eun ; 
  • Jeon, Hyongjun ; 
  • Ryu, Ka-Young ; 
  • Nam, Jin han ; 
  • Jeon, Seong Gak ; 
  • Park, HyunHee ; 
  • Lee, Ji-Soo ; 
  • Han, Kyung-Min ; 
  • et al
  • 2020-11
  • Lee, Hyun-ju. (2020-11). The novel DYRK1A inhibitor KVN93 regulates cognitive function, amyloid-beta pathology, and neuroinflammation. doi: 10.1016/j.freeradbiomed.2020.08.030
  • Elsevier Inc.
  • View : 638
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