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The Role of Increased Mitochondrial Superoxide in Pathogenesis of familial Alzheimer’s Disease through Early Neuronal Differentiation
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dc.contributor.advisor 서진수 -
dc.contributor.author Bae, Simsung -
dc.date.accessioned 2022-03-07T16:00:45Z -
dc.date.available 2022-03-07T16:00:45Z -
dc.date.issued 2022 -
dc.identifier.uri http://dgist.dcollection.net/common/orgView/200000593631 en_US
dc.identifier.uri http://hdl.handle.net/20.500.11750/16338 -
dc.description Alzheimer’s Disease (AD), human induced pluripotent stem cells (hiPSCs), mitochondrial reactive oxygen species (mtROS), neuronal differentiation, energy metabolism -
dc.description.statementofresponsibility N -
dc.format.extent 38 -
dc.language eng -
dc.publisher DGIST -
dc.subject Alzheimer’s Disease (AD), human induced pluripotent stem cells (hiPSCs), mitochondrial reactive oxygen species (mtROS), neuronal differentiation, energy metabolism -
dc.title The Role of Increased Mitochondrial Superoxide in Pathogenesis of familial Alzheimer’s Disease through Early Neuronal Differentiation -
dc.title.alternative 증가된 미토콘드리아의 초과산화물이 신경세포의 초기 분화속도 조절을 통해 가족성 알츠하이머의 발병에 미치는 영향 연구 -
dc.type Thesis -
dc.identifier.doi 10.22677/thesis.200000593631 -
dc.description.degree Master -
dc.contributor.department Brain and Cognitive Sciences -
dc.identifier.bibliographicCitation Bae, Simsung. (2022). The Role of Increased Mitochondrial Superoxide in Pathogenesis of familial Alzheimer’s Disease through Early Neuronal Differentiation. doi: 10.22677/thesis.200000593631 -
dc.contributor.coadvisor Um, Ji Won -
dc.date.awarded 2022/02 -
dc.publisher.location Daegu -
dc.description.database dCollection -
dc.citation XT.BM 배58 202202 -
dc.date.accepted 1/21/22 -
dc.contributor.alternativeDepartment 뇌인지과학전공 -
dc.embargo.liftdate 20250228 -
dc.contributor.affiliatedAuthor Bae, Simsung -
dc.contributor.affiliatedAuthor Seo, Jinsoo -
dc.contributor.affiliatedAuthor Um, Ji Won -
dc.contributor.alternativeName 배심성 -
dc.contributor.alternativeName Seo, Jinsoo -
dc.contributor.alternativeName 엄지원 -
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